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mouse anti arc (c-7)  (Santa Cruz Biotechnology)


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    Structured Review

    Santa Cruz Biotechnology mouse anti arc (c-7)
    Mouse Anti Arc (C 7), supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+arc+(c-7)/mouse+anti+arc/pm40208793-280-2-13
    Average 90 stars, based on 1 article reviews
    mouse anti arc (c-7) - by Bioz Stars, 2026-10
    90/100 stars

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    Related Articles

    other:

    Article Title: Detection of Arc/Arg3.1 oligomers in rat brain: constitutive and synaptic activity-evoked dimer expression in vivo
    Article Snippet: Primary antibodies used in this study include mouse monoclonal anti-Arc (C-7; Santa Cruz Biotechnology, Dallas, TX, Cat# sc-17839, RRID:AB_626696), rabbit polyclonal anti-Arc (Synaptic Systems, Göettingen, Germany, Cat# 156003, RRID:AB_887694), mouse anti-DJ-1 (Santa Cruz Biotechnology Cat# sc-55572, RRID:AB_831639), mouse anti-β-tubulin (Santa Cruz Biotechnology Cat# sc-166729, RRID:AB_2010699) and mouse anti-β-actin (Sigma-Aldrich Cat# A5441, RRID:AB_476744).

    Article Title: Development and Validation of Arc Nanobodies: New Tools for Probing Arc Dynamics and Function
    Article Snippet: Primary antibodies are as follows: mouse anti-Arc (C-7) (Santa Cruz Biotechnology, Dallas, TX, Cat# sc-17839, RRID:AB_626696), rabbit anti-Arc (Synaptic Systems, Göttingen, Germany, Cat# 156 003, RRID:AB_887694), rabbit anti-GFP (Santa Cruz Biotechnology Cat# sc-8334, RRID:AB_641123), mouse anti-mCherry (Takara Bio, Siga, Japan, Cat# 632,543, RRID:AB_2307319), anti-ALFA HRP-coupled sdAb (NanoTag Biotechnologies, Cat#. N1501-HRP).

    Article Title: FXR1 regulation of parvalbumin interneurons in the prefrontal cortex is critical for schizophrenia-like behaviors.
    Article Snippet: The primary antibodies used were: chicken antiGFP (A10262, Invitrogen, Carlsbad, CA), mouse antiFXR1P (1:500, 1F10A8D11D9, NeoClone), rabbit anti-FXR1P (1:500, A303-892A, Bethyl Lab), rabbit antiParvalbumin (1:5000, ab11427, Abcam), rabbit anti-c-Fos (1:1000, #2250, Cell Signaling Technology), mouse antiHA Tag (1:500, MMS-101R, Convance), mouse anti-Cav3.2 (1:500, clone N55/10, NeuroMab), rat antimCherry (1:5000, M11217, ThermoFisher), rabbit antiSST (1:200, MAB354, Sigma-Aldrich), rabbit anti- VIP (1:500, 20077, Immunostar), mouse anti- Arc (C-7) (1:500, sc-17839, Santa Cruz), rabbit anti- MYBPC1 (1:100, 22900-1-AP, Proteintech), rabbit anti- DNAH6 (1:100, 18080-1-AP, Proteintech), rabbit anti- SLC7A3 (1:500, ARP43806_P050, Aviva Systems Biology).

    Article Title: Detection of Arc/Arg3.1 oligomers in rat brain: constitutive and synaptic activity-evoked dimer expression in vivo
    Article Snippet: Primary antibodies used in this study include mouse monoclonal anti-Arc (C-7; Santa Cruz Biotechnology, Dallas, TX, Cat# sc-17839, RRID:AB_626696), rabbit polyclonal anti-Arc (Synaptic Systems, Göettingen, Germany, Cat# 156003, RRID:AB_887694), mouse antiDJ-1 (Santa Cruz Biotechnology Cat# sc-55572, RRID:AB_831639), mouse anti-β-tubulin (Santa Cruz Biotechnology Cat# sc-166729, RRID:AB_2010699) and mouse anti-β-actin (Sigma-Aldrich Cat# A5441, RRID:AB_476744).

    Article Title: Development and Validation of Arc Nanobodies: New Tools for Probing Arc Dynamics and Function.
    Article Snippet: Primary antibodies are as follows: mouse anti-Arc (C-7) (Santa Cruz Biotechnology, Dallas, TX, Cat# sc-17839, RRID:AB_626696), rabbit anti-Arc (Synaptic Systems, Göttingen, Germany, Cat# 156 003, RRID:AB_887694), rabbit anti-GFP (Santa Cruz Biotechnology Cat# sc-8334, RRID:AB_641123), mouse anti-mCherry (Takara Bio, Siga, Japan, Cat# 632,543, RRID:AB_2307319), antiALFA HRP-coupled sdAb (NanoTag Biotechnologies, Cat#. N1501-HRP).

    Incubation:

    Article Title: Binge-like Prenatal Ethanol Exposure Causes Impaired Cellular Differentiation in the Embryonic Forebrain and Synaptic and Behavioral Defects in Adult Mice
    Article Snippet: .. The blots were incubated for 3 h at room temperature or overnight at 4 °C with the following individual primary antibodies: anti-rabbit cleaved caspase-3 (CC3) (Asp175) (polyclonal, #9661, 1:1000); anti-mouse-β-actin (monoclonal, #3700, 1:1000); anti-rabbit CREB (monoclonal, #9197, 1:1000); anti-rabbit phospho-CREB (monoclonal, #9198, 1:1000); anti-rabbit p44/42 MAPK (ERK1/2) (polyclonal, # 9102, 1:2000); anti-rabbit-phospho-p44/42 MAPK (polyclonal, # 9101, 1:1000); anti-rabbit GluA1 (monoclonal, #13185) from Cell Signaling (Danvers, MA, USA); anti-mouse-Arc (C-7, #sc-17839, 1:1000); anti-mouse-Egr1 (monoclonal, S-25, #sc-101033, 1:500) from Santa Cruz Biotechnology, Inc. (Santa Cruz, CA, USA); anti-rabbit nucleoprotein expressed in proliferative cells (Ki67) (polyclonal, #PA5-19462); anti-mouse LIM homeobox 2 (Lhx2) (monoclonal, # MA5-15834) from Thermo Fisher (Waltham, MA, USA); anti-rabbit Paired Box 6 (Pax6) (polyclonal, #12323-1-AP) from Proteintech (Rosemont, IL, USA); anti-rabbit fibroblast growth factor 1 (Fgf1) (polyclonal, #ab207321) from Abcam (Waltham, MA, USA); and anti-rabbit GluN1 (polyclonal, #05-432) from Millipore Sigma (Burlington, MA, USA). ..

    Staining:

    Article Title: Defective engram allocation contributes to impaired fear memory performance in Down syndrome
    Article Snippet: Finally, samples were washed with PBS-T 0.5 % (3×15 min) followed by PBS washing (3×10 min) to remove the detergent and sections were mounted and coverslipped into a pre-cleaned glass slide with Fluoromount-G medium with DAPI (Thermo Fisher Scientific #00-4959-52). c-Fos was stained with rabbit anti-c-Fos (1:1000, Santacruz, #Sc-7202) and visualized with anti-rabbit Alexa-647 (1:500; Thermo Fisher Scientific, #A-21443). .. Arc was stained with mouse anti-Arc(C-7) (1:400; Santa Cruz, #sc-17839) and visualized with anti-mouse Alexa-488 (1:500; Thermo Fisher Scientific, #A-11001). .. CREB was stained with rabbit anti-CREB (1:1000; Millipore, #06863) and visualized with anti-mouse Alexa-555 (1:500; Thermo Fisher Scientific, #A-32732).



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    Santa Cruz Biotechnology mouse monoclonal c-7 anti-arc/arg3.1
    Intracellular mGluR5 plays a critical role in glutamate and neuronal activity-induced Arc. A, striatal neurons were pretreated with NMDA antagonist MK801 (5 μm), the AMPA/kainate receptor antagonist, 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX; 10 μm), and mGluR1 antagonist CPCCOEt (20 μm) followed by treatment with glutamate (100 μm, 1 h). Glutamate increased Arc expression that was blocked by MPEP but not LY393053. Bars indicate Arc-positive/total neurons compared with control. #, p < 0.05 versus control; n = 4, more than 2500 neurons assessed per treatment. Ctl, control. B, bath application of 15 mm KCl-induced Arc in ∼30% striatal neurons. This KCl-regulated induction was decreased ∼28% by MPEP, whereas LY395053 + KCl was not significantly different from KCl alone. Bars indicate Arc positive/total neurons expressed as the percentage of KCl treatment. More than 2000 neurons per treatment from four independent experiments were assessed for Arc immunofluorescence using the High Content Imager. #, p < 0.0001 compared with untreated control. *, p < 0.005 compared with KCl treated alone. C, the proposed model for intracellular mGluR5-mediated transcriptional activation of <t>Arc/Arg3.1</t> is shown. NPC, nuclear pore complex.
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    Santa Cruz Biotechnology mouse anti arc c 7 antibody
    Intracellular mGluR5 plays a critical role in glutamate and neuronal activity-induced Arc. A, striatal neurons were pretreated with NMDA antagonist MK801 (5 μm), the AMPA/kainate receptor antagonist, 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX; 10 μm), and mGluR1 antagonist CPCCOEt (20 μm) followed by treatment with glutamate (100 μm, 1 h). Glutamate increased Arc expression that was blocked by MPEP but not LY393053. Bars indicate Arc-positive/total neurons compared with control. #, p < 0.05 versus control; n = 4, more than 2500 neurons assessed per treatment. Ctl, control. B, bath application of 15 mm KCl-induced Arc in ∼30% striatal neurons. This KCl-regulated induction was decreased ∼28% by MPEP, whereas LY395053 + KCl was not significantly different from KCl alone. Bars indicate Arc positive/total neurons expressed as the percentage of KCl treatment. More than 2000 neurons per treatment from four independent experiments were assessed for Arc immunofluorescence using the High Content Imager. #, p < 0.0001 compared with untreated control. *, p < 0.005 compared with KCl treated alone. C, the proposed model for intracellular mGluR5-mediated transcriptional activation of <t>Arc/Arg3.1</t> is shown. NPC, nuclear pore complex.
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    Image Search Results


    Intracellular mGluR5 plays a critical role in glutamate and neuronal activity-induced Arc. A, striatal neurons were pretreated with NMDA antagonist MK801 (5 μm), the AMPA/kainate receptor antagonist, 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX; 10 μm), and mGluR1 antagonist CPCCOEt (20 μm) followed by treatment with glutamate (100 μm, 1 h). Glutamate increased Arc expression that was blocked by MPEP but not LY393053. Bars indicate Arc-positive/total neurons compared with control. #, p < 0.05 versus control; n = 4, more than 2500 neurons assessed per treatment. Ctl, control. B, bath application of 15 mm KCl-induced Arc in ∼30% striatal neurons. This KCl-regulated induction was decreased ∼28% by MPEP, whereas LY395053 + KCl was not significantly different from KCl alone. Bars indicate Arc positive/total neurons expressed as the percentage of KCl treatment. More than 2000 neurons per treatment from four independent experiments were assessed for Arc immunofluorescence using the High Content Imager. #, p < 0.0001 compared with untreated control. *, p < 0.005 compared with KCl treated alone. C, the proposed model for intracellular mGluR5-mediated transcriptional activation of Arc/Arg3.1 is shown. NPC, nuclear pore complex.

    Journal: The Journal of Biological Chemistry

    Article Title: Activation of Intracellular Metabotropic Glutamate Receptor 5 in Striatal Neurons Leads to Up-regulation of Genes Associated with Sustained Synaptic Transmission Including Arc/Arg3.1 Protein *

    doi: 10.1074/jbc.M111.301366

    Figure Lengend Snippet: Intracellular mGluR5 plays a critical role in glutamate and neuronal activity-induced Arc. A, striatal neurons were pretreated with NMDA antagonist MK801 (5 μm), the AMPA/kainate receptor antagonist, 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX; 10 μm), and mGluR1 antagonist CPCCOEt (20 μm) followed by treatment with glutamate (100 μm, 1 h). Glutamate increased Arc expression that was blocked by MPEP but not LY393053. Bars indicate Arc-positive/total neurons compared with control. #, p < 0.05 versus control; n = 4, more than 2500 neurons assessed per treatment. Ctl, control. B, bath application of 15 mm KCl-induced Arc in ∼30% striatal neurons. This KCl-regulated induction was decreased ∼28% by MPEP, whereas LY395053 + KCl was not significantly different from KCl alone. Bars indicate Arc positive/total neurons expressed as the percentage of KCl treatment. More than 2000 neurons per treatment from four independent experiments were assessed for Arc immunofluorescence using the High Content Imager. #, p < 0.0001 compared with untreated control. *, p < 0.005 compared with KCl treated alone. C, the proposed model for intracellular mGluR5-mediated transcriptional activation of Arc/Arg3.1 is shown. NPC, nuclear pore complex.

    Article Snippet: Primary antibodies included polyclonal anti-C-terminal mGluR5 (1:250; Millipore), mouse monoclonal C-7 anti-Arc/Arg3.1 (1:100; Santa Cruz Biotechnology), rabbit polyclonal anti-SRF (1:1000, Santa Cruz), anti-NeuN (1:100, Millipore), monoclonal anti-lamin B 2 (1:100; Zymed Laboratories Inc.), polyclonal anti-EAAT3 (1:1000; Chemicon International, Temecula, CA), polyclonal anti-quisqualate antibody (1:250; Dr. J. Koerner, University of Minnesota), and polyclonal anti-pERK1/2 (1:200; Cell Signaling Technology, Beverly, MA).

    Techniques: Activity Assay, Expressing, Immunofluorescence, Activation Assay